As shown in Figure 7A, there was a significant enhancement in the H2-K1 signaling pathway and a notable weakening of the THBS-1 signaling pathway between tumor cells and CD8+T cells following RBMX knockout; the expression of H2-K1 in tumor cells significantly increased, while the THBS-1 expression was decreased (Figure 7A). This evidence concerns the gene RBMX and neoplasm.