Numerous studies have demonstrated that μg can disrupt cell tensegrity, reorganize the cytoskeleton, suppress pro-survival pathways such as NF-κB, and activate key pro-apoptotic mediators including p53, Bax, FAS, and Caspase-8, ultimately impairing the viability and proliferative capacity of tumor cells [95,149]. This evidence concerns the gene NFKB1 and neoplasm.