These hiPSC-derived cardiomyocytes (hiPSC-CMs) exhibit a functional dystrophin–glycoprotein complex (DGC) localized to the membrane, show electrophysiological abnormalities and arrhythmogenicity, and reproduce phenotypes observed in patients with DMD cardiomyopathy [13,18,19]. This evidence concerns the gene DMD and cardiomyopathy.