Salivary effectors of insect vectors can enhance the post‐transmission infections of insect‐borne viruses, including cell‐to‐cell movement and long‐distance phloem spread.[10, 31] For example, salivary protein carbonic anhydrase II of M. persicae facilitates cell‐to‐cell translocation by accelerating intercellular vesicle trafficking,[31] while GLD promotes the formation of highly ordered aggregated SEO, which in turn promotes the systemic movement of CMV by interacting with CMV CP. This evidence concerns the gene CA2 and infection.