Moreover, both MDMs, MDDCs, and Mon subsets induced high expression of STAT1-dependent chemokines, including CCL2, CCL5, CCL8, CXCL10, and/or CXCL11 (Fig 6D) which play a key role in the recruitment of different immune cell populations at the site of infections, including Mon, NK cells, T cells, and B cells, leading to the amplification of innate and adaptive immune responses [80,81]. This evidence concerns the gene STAT1 and infection.