The most common types of amyloidosis include AL amyloidosis, characterized by the production of light chains associated with monoclonal plasma cell populations; transthyretin (TTR) amyloidosis, where normal TTR aggregates predominantly causing cardiomyopathy; and AA amyloidosis, which arises from the deposition of acute phase reactant serum amyloid A (SAA) protein, that is, a highly conserved precursor synthesized by hepatocytes in systemic and inflammatory diseases [4, 5]. Here, TTR is linked to AA amyloidosis.