IL6 and cystic fibrosis: In air‐liquid interface (ALI) cultures of primary human bronchial epithelia derived from normal donors or cystic fibrosis (CF) patients, ORP‐100 attenuated saline‐stimulated release of pro‐inflammatory cytokines tumor necrosis factor alpha (TNF‐α; Figure 1D) and interleukin‐6 (IL‐6; not shown) to a significantly greater extent than did rhTRX.