Taken together, our results suggest that BBR‐CA could function by modulating the PI3K/AKT/mTOR signaling pathway, resulting in decreased nuclear expression of SREBP‐1, as well as reduced expression of stearoyl‐CoA desaturase 1 and acetyl‐CoA carboxylase, thus alleviating hyperlipidemia. The gene discussed is MTOR; the disease is hyperlipidemia.