PROS1 and myalgic encephalomeyelitis/chronic fatigue syndrome: This was made possible by PFS offspring having normal‐functioning mitochondria, as evidenced by comparable levels of mitochondrial function–related proteins involved in biogenesis (Figure 8a,b), electron transport chain enzymes (Figure 8a,c), fusion/fission proteins (Figure 8a,d) and mitophagy proteins (Figure 8a,e) compared with CFS offspring.