NFKBIA and myocarditis: Following the accumulation of neutrophils at the site of myocarditis injury,neutrophil TLRs recognize myocardial infection, degrade NF-κB inhibitorα (IκB-α), and activate the secretory vesicle pathway,Golgi–endoplasmic reticulum pathway, and cell lysis pathway implicated incytokine release upon neutrophil activation [14].