respiratory disease dataset, we observed significant enrichment of IFNγ signatures in cystic fibrosis (CF) and COVID‐19 infection, IL‐33 in COPD and COVID‐19, IL‐4 + IL‐13 in pneumonia, and TGFβ signatures in pulmonary fibrosis (PF) and chronic rhinosinusitis (CRS) (Figure 6). This evidence concerns the gene IL4 and susceptibility to pneumonia measurement.