The findings suggested that Flt3L, MCP2, and MCP4 may be predisposing factors in the initiation of PCa, while adenosine deaminase, axin-1, CXCL6, IL-24, IL-33, and Flt3L in a downstream position in the pathogenesis of PCa. The gene discussed is AXIN1; the disease is posterior cortical atrophy.