The results suggested a causal relationship between fms-related tyrosine kinase 3 ligand (Flt3L), recombinant monocyte chemotactic protein (MCP) 2, MCP4, and the incidence of PCa (odds ratio [OR]: 1.0016, 95% confidence interval [CI]: 1.0000–1.0032, P = .045; OR: 0.9979, 95% CI: 0.9958–1.0000, P = .045; OR: 1.0012, 95% CI: 1.0001–1.0023, P = .031). This evidence concerns the gene FLT3LG and posterior cortical atrophy.