Notably, Slc7a11 exhibits unique biological functions in malaria parasite liver‐stage infection regulation: targeted inhibition of GPX4 or SLC7A11 significantly suppresses parasite proliferation through enhanced LPO, whereas NOX1 (NADPH oxidase 1, NADPH1) and TFR1 blockade produces opposing effects [395]. The gene discussed is NOX1; the disease is malaria.