It drives tumor progression through multiple mechanisms: UCA1 can act as a molecular sponge for tumor-suppressive miRNAs, it modulates signaling pathways like mTOR and STAT3 to enhance glycolysis and proliferation, and it interacts with epigenetic regulators to alter gene expression (Zhang et al., 2019; Li et al., 2014b). The gene discussed is MTOR; the disease is neoplasm.