Network analysis and molecular docking predicted seven putative targets, with enrichment analyses revealing these targets’ modulation of key AMD-associated pathological pathways—including angiogenesis (e.g., VEGFA pathway), apoptosis (e.g., BCL2 pathway), and extracellular matrix metabolism (e.g., MMP2/MMP9 pathway). The gene discussed is BCL2; the disease is age-related macular degeneration.