This reduces the clearance of translocated E. faecalis, worsening liver disease.[26] Additionally, studies indicate that KCs undergo necroptosis after severe Listeria infection, while MoMFs enhance antibacterial immunity through IFN‐γ‐mediated responses.[27] Using a wild‐type mouse model (Figure 5), we investigated how S. saprophyticus and T‐2 toxin dynamically disrupt hepatic macrophage homeostasis and contribute to liver disease progression. Here, TBXT is linked to listeriosis.