Specifically, the Intermediate group had upregulated genes associated with neurodevelopmental disorders (e.g., Shank1‐3) whereas the Susceptible group was more genetically distinct from the Resistant rats, with upregulation of genes related to synaptic function (e.g., Camk2a) and downregulation of genes involved in satiety (e.g., Cck), inflammatory processes (e.g., Ccl27), and neuronal growth (e.g., Bdnf). Here, SHANK1 is linked to neurodevelopmental disorder.