Decreased levels of miR-214-3p have been demonstrated to protect renal TECs from ferroptosis and alleviate tubular damage in cisplatin-induced AKI, as evidenced in both cellular and animal models, by upregulating GPX4 and solute carrier family 7, member 11(SLC7A11) while downregulating Acyl-CoA synthetase long-chain family member 4(ACSL4) expression [160]. This evidence concerns the gene GPX4 and acute kidney injury.