PON1 and triple-A syndrome: In fact, endogenous antioxidantdefenses [such as Cu/Zn and Mn superoxide dismutase, glutathione reductaseand glutathione peroxidase,−,  Paraoxonase-1, Nrf2/Heme-oxygenase 1 andGlutathione-SH] were shown to be dysregulatedin AAA and PAD, as described in a recent review. Moreover, our results agree with the ones previously reportedfor atherosclerotic AAA and thoracic AAA (atherosclerotic and nonatherosclerotic),where the authors found reduced levels of PE plasmalogens in aortictissue samples.