TCF4 and myocardial infarction: Increasing PBMSC‐Exos production, enhancing CMs biogenesis, or activating β‐catenin target genes (particularly Tcf4) (Figure 10O,Q) may be novel, effective therapeutic avenues to improve PBMSC‐Exos‐mediated beneficent communication between RAs and MI hearts after RD, enhancing cardiac regeneration in acute MI (Figure S8).