Similarly, the levels of CCL2, IL-6, interferon−γ (IFN-γ), eotaxin, macrophage inflammatory protein−1 α (MIP-1α), IL-4, granulocyte−colony–stimulating factor (G-CSF), and CXCL10 in the vitreous of patients with high myopia with macular holes were significantly higher than those in patients with non-high myopia macular holes (78, 79). The gene discussed is IFNG; the disease is macular holes.