After sodium octanoate intervention, the expression levels of Claudin1, ZO-1, and IL-10 were restored to varying degrees, especially IL-10 in the CA group even exceeded that of the control group, possibly reflecting its compensatory regulatory effect on inflammation; while the expression of IL-6 and IL-17 significantly decreased, further supporting the potential role of sodium octanoate in alleviating intestinal inflammation. The gene discussed is IL6; the disease is inflammation.