STUB1 and acute kidney injury: Clonal haematopoiesis of indeterminate potential-specific mutations was visualized at the RNA level using custom mutaFISHTM probes, identifying these cells as macrophages present in inflammatory shoulder regions of coronary and carotid plaques, supporting the hypothesis that CHIP contributes to plaque instability via macrophage-driven inflammation.67 These findings align with reports linking CHIP to increased monocyte-driven inflammation and tissue damage in other disease contexts, including acute kidney injury.6