Moreover, ectopic expression of p65 rescued the alteration of NF‐κB signaling, p65 enrichment and activity, FLCN and FNIP1 expression, lysosomal biogenesis, and CTSD activity in NB cells stably over‐expressing RORB (Figure S6A–E, Supporting Information). This evidence concerns the gene NFKB1 and neuroblastoma.