CD8A and infection: Since the innate NK cell‐derived IFN‐γ in Rag2−/− mice cannot fully compensate for the CD8+ T cell‐derived IFN‐γ, one can argue that these two bursts of IFN‐γ may be functionally different, and further studies are needed to fully characterise the importance of IFN‐γ at different times post‐infection in both viral control and immunopathology.