2021). Clinical studies and meta‐analyses have highlighted SA's efficacy in improving motor function and reducing neurological deficits in stroke patients (Y. J. Huang, Huang, et al. 2021). Mechanistic investigations further reveal that SA exerts its neuroprotective effects by activating the p62/Keap1/NRF2 signaling pathway and modulating ferroptosis‐related proteins including ferritin heavy chain 1 (FTH1) and GPX4 (M. Y. Li et al. 2022). The gene discussed is FTH1; the disease is stroke disorder.