Comparing oncolysates from rVSV- and rVSV-NDV-infected tumor cells, we observed a significantly greater upregulation of MHC-I, MHC-II, and CD86 on DCs that had been co-cultured with oncolysates from syncytia-forming rVSV-NDV (Figures 7J and 7K). The gene discussed is CD86; the disease is neoplasm.