SMARCB1 and rhabdoid tumor: Leruste et al. reported that the immunogenicity of rhabdoid tumors, measured by the cytolytic activity (CYT) score20, is mediated by the re-expression of endogenous retroviruses and the activation of interferon signaling, arising from dysfunctional chromatin regulation as a result of SMARCB1 loss14, but previously we did not observe epigenetic derepression of retroviral elements15.