TARDBP and proteostasis deficiencies: GFP‐fusion constructs lacking RRM1 (residues 106–175), RRM2 (residues 193–257) or a C‐terminal region (residues 257–414) failed to produce any uncoordinated phenotype, as well as a TDP‐43 construct with an inactivated nuclear localisation signal (that restricts its localisation strictly to the cytosol), in contrast to the full‐length or other fusion TDP‐43 deletion constructs (Table 3 enlists the TDP‐43 construct variants used to study TDP‐43 proteinopathies in C. elegans).