PPARA and diabetes mellitus: Accumulating information indicates that MD, although not significantly contrasting diabetes‐induced down‐regulation of the molecular PPARα‐CPT1 axis, one of the major upstream controllers of mitochondrial fat oxidation, was able to qualitatively/quantitatively restore the liver pool of acylated carnitine derivatives, suggesting normalized fatty acid flux toward mitochondria, β‐oxidation, as well as preserved hepatic response to hypoxia [60, 61].