Cyclin-dependent kinases, such as CDK7 and CDK9, are functionally connected to the processes of transcription initiation and elongation in various tumors and facilitate the malignant proliferation of tumor cells.79–81 In the present study, we determined the regulatory pattern of CDKs in downstream tumor proliferation-related molecules and elucidated the biological mode of uncontrolled growth of primary tumor cells. The gene discussed is CDK7; the disease is neoplasm.