In addition to the aforementioned individuals, one report of a 2‐month‐old infant with compound heterozygous variants in NARS2 (a heterozygous deletion of exons 8–9 and the p.(Pro214Leu) missense variant20) mentioned hyperglycaemia in the neonatal period and other features indicative of a mitochondrial disorder, including lactic acidosis. This evidence concerns the gene NARS2 and lactic acidosis.