Further phenotypic analysis of therapeutic cells infiltrating the kidney tumor revealed that AlloCAR70-NKT cells exhibited a more activated and cytotoxic phenotype, characterized by elevated expression of activation markers (i.e., CD25 and CD69), pro-inflammatory cytokines (i.e., IFN-γ), and cytotoxic effectors (i.e., Granzyme B and Perforin) (Figures 4L and 4M). This evidence concerns the gene PRF1 and kidney neoplasm.