GAP43 and ischemic stroke: Growth-associated protein 43 (GAP-43) and neurofilament 200 (NF-200) promote axonal regeneration and synaptic plasticity, while paired immunoglobulin-like receptor B (PirB) inhibits neuronal burst growth by activating RhoA, thereby suppressing motor function recovery after ischemic stroke (Deng et al., 2018).