In our research, tead1a exhibited a promoting effect on neutrophils at an earlier stage of granulocytic development, and its mechanism of action was independent of G‐CSF (csf3r in zebrafish).[53, 54] Moreover, there have been no reports of myeloid malignancies with high expression of the tead1 gene, which theoretically holds major research value and clinical implications. The gene discussed is TEAD1; the disease is myeloid neoplasm.