TP53 and oral lichen planus: Therefore, it has been suggested that enhanced expression of p53 protein might be secondary to cellular stress due to inflammatory conditions in OLP (Ögmundsdóttir et al. 2002) or may present a physiological response to the hyperproliferative state of OLP (Schifter et al. 1998) and also, is also induced by wild‐type p53, which tries to arrest the cell cycle in response to DNA damage (Engeland 2022).