CD8A and neoplasm: found that DCs in the TME are a major source of PD‐L1, despite being outnumbered by PD‐L1+ macrophages, and that selectively deleting PD‐L1 from DCs, but not macrophages, boosts anti‐tumor CD8+ T cell responses and inhibits tumor growth.[166] Concordantly, Dammeijer et al.