Several EBV latency proteins have been identified as interacting with components of the mTOR pathway, including LMP1’s effect on the phosphorylated forms of mTOR, P70S6K, and 4E-BP1, as well as on Glut-1 transcription via the mTORC1/NF-κB signaling pathway to promote growth in NPC tumors [42,43]. The gene discussed is PDLIM7; the disease is nasopharyngeal carcinoma.