Its overexpression was associated with decreased levels of ovalbumin-specific IgE, IL-4, IL-5, and IL-13, contributing to improved asthma outcomes by limiting airway remodeling and autophagy through the downregulation of matrix metalloproteinase (MMP)-16 and autophagy related 7 (ATG7) [63]. The gene discussed is IL13; the disease is asthma.