SOAT1 and Hyperglycemia: Thus, the results substantiate the mechanism whereby these core components (e.g., bornyl acetate, (−)-spathulenol, and (−)-pogostol) mitigate hyperglycemia and oxidative stress by regulating pivotal targets (e.g., CYP3A4, PPARA, JAK2) to activate a multidimensional network involving (e.g., the PPAR signaling pathway, nuclear receptor-mediated steroid hormone pathway, and JAK-STAT signaling pathway).