These include autoantibodies directed against angiotensin-converting enzyme 2 (ACE2) [95], β2-adrenergic receptors, muscarinic M2 acetylcholine receptors, angiotensin II type 1 receptors (AT1R), and the Mas receptor involved in angiotensin-(1–7) signaling [96], suggesting possible autoimmune mechanisms contributing to PASC. This evidence concerns the gene ACE2 and long COVID-19.