Accordingly, sustained stimulation of the angiotensin II type 1 receptor (AT1R) has been implicated in the pathophysiology of long-term sequelae following SARS-CoV-2 infection (long COVID), potentially contributing to adverse effects such as vasoconstriction, elevated blood pressure, chronic inflammation, oxidative stress, cardiac hypertrophy, fibrosis affecting multiple organs (including the heart, lungs, kidneys, and liver), as well as sensory impairments (such as anosmia and ageusia) and neurological dysfunctions [8,61]. This evidence concerns the gene AGTR1 and Ageusia.