STK32C, a member of the AGC family of kinases has been implicated in HMGB1 signaling in bladder cancer progression [56] and was recently shown to be involved in mediating resistance to doxorubicin in TNBC cells [57], while STK40, a pseudokinase, was shown to be involved in mesoderm development, migration of endothelial cells and, of relevance here, in promoting the growth of triple negative breast cancer cells [58,59,60]. This evidence concerns the gene STK32C and urinary bladder cancer.