After 7 days of NLX administration, we did not observe any significant changes in either atherosclerotic plaque localized in aortic arches (NaCl 21.30% ± 9.04 vs. NLX 26.21% ± 9.22) (F[(1, 12) = 1.01; p = 0.33]) (Figure 2a) or liver steatosis (NaCl 2.46% ± 3.51 vs. NLX 1.47% ± 1.95) (F[(1, 10) = 0.36; p = 0.56]) in the ApoE−/− mice (Figure 2b). This evidence concerns the gene APOE and fatty liver disease.