Clinical studies examining serum cytokine levels have identified COVID-19 hyperinflammation as being characterized by significantly elevated concentrations of a range of cytokines, including G-CSF, HGF, IFN-α2, IFN-γ, IL-10, IL-12, IL-17, IL-1ra, IL-1α, IL-1β, IL-2, IL-2R, IL-4, IL-6, IL-7, IL-8, IP-10, MCP-1, M-CSF, MIP-1α, MIP-3α (herein referred to as CCL20), MIP-3β, PDGF-BB, and TNF [7,8,9,10,11]. This evidence concerns the gene CCL20 and COVID-19.