This event primes dormant cancer cells with dormancy-surviving programmes, such as autophagy [112], stress-induced p38-regulated transcription factor network [69], EMT programme [104, 113], DNA damage repair [114], and NR2F1-mediated quiescence programmes [109], which are key to the survival, invasiveness, and treatment-resistant phenotype of these cells. The gene discussed is NR2F1; the disease is cancer.