MID1IP1 and hepatocellular carcinoma: Furthermore, researchers studied the oncogenic potential of MID1IP1 in hepatocellular carcinoma cell (HCC) growth in relation to ribosomal protein L5 and L11 and CNOT2-mediated c-Myc signaling and found that CNOT2 knockdown could further inhibit the downregulation of MID1IP1 by c-Myc.