The study concluded that differential alternative splicing of Amyloid beta A4 protein (APP)/Serine/threonine-protein kinase 16 (STK16) is associated with ELK1’s diverse roles in gene transcription and may be of great interest in the context of ELK1 implications in pathophysiology of diseases like glioma [321]. This evidence concerns the gene ELK1 and central nervous system cancer.