Furthermore, in vivo experiments revealed the presence of oligomeric amyloid precursor protein‐derived C‐terminal fragments (APP‐CTFs) in AD mouse models, the levels of which are selectively enriched in endolysosomal compartments, including EVs, and amplified by γ‐secretase inhibition (Lauritzen et al. 2019) (Table 1). The gene discussed is APP; the disease is Alzheimer disease.