Induction of tumorigenic attributes (increased wound healing, migration and invasion capabilities) in both cell lines.Induction of oncogenes (P110α, Akt, mTOR, NFKB1 and RAF).Attenuation of tumor suppressor (TS) genes in Cd-exposed RWPE1 cells.Enrichment of prostate cancer related pathways cells exposed to Cd. This evidence concerns the gene NFKB1 and prostate carcinoma.