Among cancer patients, almost EGFR mutations are classical, either exon 19 deletions (Ex19Del) and/or L858R with or without T790M, while atypical EGFR mutations occur primarily in exons 18‐20, such as G719X and L861Q.[104] The underlying molecular mechanisms and the functional implications of these non‐canonical characteristics of mutant EGFR warrant further investigation in future research. Here, EGFR is linked to cancer.